Medical Physiology, 3rd Edition

References

Books and Reviews

Cherniack NS, Longobardo GS. Abnormalities in respiratory rhythm. American Physiological Society: Bethesda, MD; 1986:729–749. Cherniack NS, Widdicombe J. Handbook of Physiology, Section 3: The Respiratory System. vol 2.

Coleridge JCG. Pulmonary reflexes: Neural mechanisms of pulmonary defense. Annu Rev Physiol. 1994;56:69–91.

Feldman JL. Neurophysiology of breathing in mammals. American Physiological Society: Bethesda, MD; 1986:463–525. Bloom FE. Handbook of Physiology, Section 1: The Nervous System. vol 4.

Loeschcke HH. Central chemosensitivity and the reaction theory. J Physiol. 1982;332:1–24.

Peers C, Buckler KJ. Transduction of chemostimuli by the Type I carotid body cell. J Membr Biol. 1995;144:1–9.

Richerson GB. Serotonergic neurons as carbon dioxide sensors that maintain pH homeostasis. Nature Rev Neurosci. 2004;5:449–461.

Journal Articles

Fencl V, Miller TB, Pappenheimer JR. Studies on the respiratory response to disturbances of acid-base balance, with deductions concerning the ionic composition of cerebral interstitial fluid. Am J Physiol. 1966;210:459–472.

Gray PA, Janczewski WA, Mellen N, et al. Normal breathing requires pre-Bötzinger complex neurokinin-1 receptor-expressing neurons. Nat Neurosci. 2001;4(9):1–4.

Pappenheimer JR, Fencl V, Heisey SR, Held D. Role of cerebral fluids in control of respiration as studied in unanesthetized goats. Am J Physiol. 1965;208:436–450.

Ptak K, Yamanishi T, Aungst J, et al. Raphé neurons stimulate respiratory circuit activity by multiple mechanisms via endogenously released serotonin and substance P. J Neurosci. 2009;29:3720–3737.

Schläfke ME, See WR, Loeschcke HH. Ventilatory response to alterations of H+ ion concentration in small areas of the ventral medullary surface. Resp Physiol. 1970;10:198–212.

Wang W, Zaykin AV, Tiwari JK, et al. Acidosis-stimulated neurons of the medullary raphe are serotonergic. J Neurophysiol. 2001;85:2224–2235.

Williams SEJ, Wootton P, Mason HS, et al. Hemoxygenase-2 is an oxygen sensor for a calcium-sensitive potassium channel. Science. 2004;206:2093–2097.